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Conference Paper: Loss of function in toll-like receptor 4 receptors nitric oxide-mediated relaxations in LEPR(vdb/db) mice

TitleLoss of function in toll-like receptor 4 receptors nitric oxide-mediated relaxations in LEPR(vdb/db) mice
Authors
KeywordsCardiovascular disease
Issue Date2010
PublisherHong Kong College of Cardiology. The Journal's web site is located at http://www.hkcchk.com/journals.php#3
Citation
The 14th Annual Scientific Meetingof the Institute of Cardiovascular Science and Medicine, Hong Kong, China, 18 December 2010. In Journal of the Hong Kong College of Cardiology, 2010, v. 18 n. 2, p. 63, abstract no. OC1 How to Cite?
AbstractThe present study analyzes the role of toll-like receptor 4 (TLR4), a target for saturated fatty acids and lipopolysaccharides, in modulating endothelial function in the mouse aorta. A type-2 diabetes model with double knockout (DKO) of leptin receptors (Lepr) and TLR4, was obtained by crossing Leprdb/+ and TLR4-/- mice. Mice were sacrificed and ...
DescriptionThis journal issue is proceedings of ICSM 2010
Abstracts for Oral Communications
Persistent Identifierhttp://hdl.handle.net/10722/136053
ISSN
2015 SCImago Journal Rankings: 0.102

 

DC FieldValueLanguage
dc.contributor.authorLiang, Cen_US
dc.contributor.authorXu, Aen_US
dc.contributor.authorVanhoutte, PMen_US
dc.date.accessioned2011-07-27T02:02:14Z-
dc.date.available2011-07-27T02:02:14Z-
dc.date.issued2010en_US
dc.identifier.citationThe 14th Annual Scientific Meetingof the Institute of Cardiovascular Science and Medicine, Hong Kong, China, 18 December 2010. In Journal of the Hong Kong College of Cardiology, 2010, v. 18 n. 2, p. 63, abstract no. OC1en_US
dc.identifier.issn1027-7811-
dc.identifier.urihttp://hdl.handle.net/10722/136053-
dc.descriptionThis journal issue is proceedings of ICSM 2010-
dc.descriptionAbstracts for Oral Communications-
dc.description.abstractThe present study analyzes the role of toll-like receptor 4 (TLR4), a target for saturated fatty acids and lipopolysaccharides, in modulating endothelial function in the mouse aorta. A type-2 diabetes model with double knockout (DKO) of leptin receptors (Lepr) and TLR4, was obtained by crossing Leprdb/+ and TLR4-/- mice. Mice were sacrificed and ...-
dc.languageengen_US
dc.publisherHong Kong College of Cardiology. The Journal's web site is located at http://www.hkcchk.com/journals.php#3-
dc.relation.ispartofJournal of the Hong Kong College of Cardiologyen_US
dc.subjectCardiovascular disease-
dc.titleLoss of function in toll-like receptor 4 receptors nitric oxide-mediated relaxations in LEPR(vdb/db) miceen_US
dc.typeConference_Paperen_US
dc.identifier.emailLiang, C: cfliang@hku.hken_US
dc.identifier.emailXu, A: amxu@hkucc.hku.hken_US
dc.identifier.emailVanhoutte, PM: vanhoutt@hku.hk-
dc.identifier.authorityXu, A=rp00485en_US
dc.identifier.authorityVanhoutte, PM=rp00238en_US
dc.description.naturelink_to_OA_fulltext-
dc.identifier.hkuros186247en_US
dc.identifier.volume18en_US
dc.identifier.issue2-
dc.identifier.spage63en_US
dc.identifier.epage63en_US
dc.publisher.placeHong Kong-
dc.description.otherThe 14th Annual Scientific Meetingof the Institute of Cardiovascular Science and Medicine, Hong Kong, China, 18 December 2010. In Journal of the Hong Kong College of Cardiology, 2010, v. 18 n. 2, p. 63, abstract no. OC1-

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