Conference Paper: SARS-CoV S protein induces strong mucosal immune responses and provides long-term protection against SARS-CoV infection
| Title | SARS-CoV S protein induces strong mucosal immune responses and provides long-term protection against SARS-CoV infection |
|---|---|
| Authors | Zheng, B Du, L Zhao, G Lin, Y Sui, H Chan, CS He, Y Jiang, S Zhou, Y Yuen, KY |
| Issue Date | 2010 |
| Publisher | Portland Press Ltd.. The Journal's web site is located at http://www.cellbiolint.org/cbi/default.htm |
| Citation | The 8th International Symposium on Frontiers in Life Sciences (生命科學前沿國際研討會 2010), YinChuan, China, 25-28 August 2010. In Cell Biology International, 2010, v. 34 n. 8, p. S62 [How to Cite?] |
| Abstract | We have previously reported that a subunit protein vaccine based on receptor binding domain (RBD) of SARS-CoV S protein and a recombinant adeno-associated virus (rAAV)-based RBD (RBD-rAAV) vaccine could induce highly potent neutralizing antibody (NA) responses in immunized animals. In this study, systemic, mucosal and cellular immune responses and long-term protective immunity induced by RBD-rAAV were further characterized in a BALB/c mouse model, with comparison of the intramuscular (i.m.) and intranasal (i.n.) routes of administration. Our results demonstrated that: (1) the i.n. vaccination induced systemic humoral immune response of comparable strength and shorter duration than the i.m. vaccination, but local humoral immune response was much stronger; (2) the i.n. vaccination elicited stronger systemic and local specific cytotoxic T cell responses than the i.m. vaccination, as evidenced by higher prevalence of IL-2 and/or IFN-c producing CD3+/CD8+ T cells in both lungs and spleen; (3) the i.n. vaccination induced a similar protection as the i.m. vaccination against SARS-CoV challenge in mice; (4) higher titers of mucosal IgA and serum NA were associated with lower viral load and less pulmonary pathological damage, while no antibody-mediated disease enhancement effect was observed; and (5) the vaccination could provide long-term protection against SARS-CoV infection. Taken together, our findings suggest that RBD-rAAV can be further developed into a vaccine candidate for prevention of SARS and that i.n. vaccination may be the preferred route of administration due to its ability to induce SARS-CoV-specific systemic and mucosal immune responses and its better safety profile. |
| Description | Theme: New Advances and Challenges in Multidisciplinary Research 主題: 多學科交叉研究的新進展與挑戰 |
| ISSN | 1065-6995 2011 Impact Factor: 1.482 2011 SCImago Journal Rankings: 0.166 |
| dc.contributor.author | Zheng, B |
|---|---|
| dc.contributor.author | Du, L |
| dc.contributor.author | Zhao, G |
| dc.contributor.author | Lin, Y |
| dc.contributor.author | Sui, H |
| dc.contributor.author | Chan, CS |
| dc.contributor.author | He, Y |
| dc.contributor.author | Jiang, S |
| dc.contributor.author | Zhou, Y |
| dc.contributor.author | Yuen, KY |
| dc.date.accessioned | 2011-07-27T02:00:05Z |
| dc.date.available | 2011-07-27T02:00:05Z |
| dc.date.issued | 2010 |
| dc.description.abstract | We have previously reported that a subunit protein vaccine based on receptor binding domain (RBD) of SARS-CoV S protein and a recombinant adeno-associated virus (rAAV)-based RBD (RBD-rAAV) vaccine could induce highly potent neutralizing antibody (NA) responses in immunized animals. In this study, systemic, mucosal and cellular immune responses and long-term protective immunity induced by RBD-rAAV were further characterized in a BALB/c mouse model, with comparison of the intramuscular (i.m.) and intranasal (i.n.) routes of administration. Our results demonstrated that: (1) the i.n. vaccination induced systemic humoral immune response of comparable strength and shorter duration than the i.m. vaccination, but local humoral immune response was much stronger; (2) the i.n. vaccination elicited stronger systemic and local specific cytotoxic T cell responses than the i.m. vaccination, as evidenced by higher prevalence of IL-2 and/or IFN-c producing CD3+/CD8+ T cells in both lungs and spleen; (3) the i.n. vaccination induced a similar protection as the i.m. vaccination against SARS-CoV challenge in mice; (4) higher titers of mucosal IgA and serum NA were associated with lower viral load and less pulmonary pathological damage, while no antibody-mediated disease enhancement effect was observed; and (5) the vaccination could provide long-term protection against SARS-CoV infection. Taken together, our findings suggest that RBD-rAAV can be further developed into a vaccine candidate for prevention of SARS and that i.n. vaccination may be the preferred route of administration due to its ability to induce SARS-CoV-specific systemic and mucosal immune responses and its better safety profile. |
| dc.description.nature | link_to_OA_fulltext |
| dc.description | Theme: New Advances and Challenges in Multidisciplinary Research |
| dc.description | 主題: 多學科交叉研究的新進展與挑戰 |
| dc.description.other | The 8th International Symposium on Frontiers in Life Sciences (生命科學前沿國際研討會 2010), YinChuan, China, 25-28 August 2010. In Cell Biology International, 2010, v. 34 n. 8, p. S62 |
| dc.identifier.citation | The 8th International Symposium on Frontiers in Life Sciences (生命科學前沿國際研討會 2010), YinChuan, China, 25-28 August 2010. In Cell Biology International, 2010, v. 34 n. 8, p. S62 [How to Cite?] |
| dc.identifier.epage | S62 |
| dc.identifier.hkuros | 188731 |
| dc.identifier.issn | 1065-6995 2011 Impact Factor: 1.482 2011 SCImago Journal Rankings: 0.166 |
| dc.identifier.issue | 8 |
| dc.identifier.spage | S62 |
| dc.identifier.uri | http://hdl.handle.net/10722/135932 |
| dc.identifier.volume | 34 |
| dc.language | eng |
| dc.publisher | Portland Press Ltd.. The Journal's web site is located at http://www.cellbiolint.org/cbi/default.htm |
| dc.publisher.place | United Kingdom |
| dc.relation.ispartof | Cell Biology International |
| dc.rights | The final version of record is available at [Journal URL]. |
| dc.title | SARS-CoV S protein induces strong mucosal immune responses and provides long-term protection against SARS-CoV infection |
| dc.type | Conference_Paper |

