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Conference Paper: Foxp3 mRNA expression on DC subsets in patients of systemic lupus erythematosus

TitleFoxp3 mRNA expression on DC subsets in patients of systemic lupus erythematosus
Authors
KeywordsMedical sciences
Rheumatology
Issue Date2010
PublisherWiley-Blackwell Publishing Asia. The Journal's web site is located at http://www.blackwell-synergy.com/loi/ijrd
Citation
The 14th Congress of Asia Pacific League of Associations for Rheumatology, Hong Kong, China, 11-15 July 2010. In International Journal of Rheumatic Diseases, 2010, v. 13 suppl. 1, p. 55, abstract no. 0557 How to Cite?
AbstractBACKGROUND: Dendritic cells (DCs), professional antigen presenting cells which link the innate and adaptive immune systems, play an important role in inducing immunity and tolerance. Systemic lupus erythematosus (SLE), an autoimmune disease with loss of tolerance to self antigens, is suggested to have apoptotic cells as the source of antigens. Foxp3, a transcription factor, is suggested to be …
DescriptionBasic and Translational Research: 1.7. Systemic lupus erythematosus, Sjögren's, systemic vasculitis and antiphospholipid syndrome - pathogenesis and animal models
Persistent Identifierhttp://hdl.handle.net/10722/129831
ISSN
2014 Impact Factor: 1.469
2014 SCImago Journal Rankings: 0.467

 

DC FieldValueLanguage
dc.contributor.authorJin, Oen_US
dc.contributor.authorKavikondala, Sen_US
dc.contributor.authorMok, MYen_US
dc.contributor.authorGu, JRen_US
dc.contributor.authorSun, LYen_US
dc.contributor.authorFu, Ren_US
dc.contributor.authorChan, Aen_US
dc.contributor.authorYeung, Jen_US
dc.contributor.authorNie, Yen_US
dc.contributor.authorLau, CSen_US
dc.date.accessioned2010-12-23T08:42:44Z-
dc.date.available2010-12-23T08:42:44Z-
dc.date.issued2010en_US
dc.identifier.citationThe 14th Congress of Asia Pacific League of Associations for Rheumatology, Hong Kong, China, 11-15 July 2010. In International Journal of Rheumatic Diseases, 2010, v. 13 suppl. 1, p. 55, abstract no. 0557en_US
dc.identifier.issn1756-1841-
dc.identifier.urihttp://hdl.handle.net/10722/129831-
dc.descriptionBasic and Translational Research: 1.7. Systemic lupus erythematosus, Sjögren's, systemic vasculitis and antiphospholipid syndrome - pathogenesis and animal models-
dc.description.abstractBACKGROUND: Dendritic cells (DCs), professional antigen presenting cells which link the innate and adaptive immune systems, play an important role in inducing immunity and tolerance. Systemic lupus erythematosus (SLE), an autoimmune disease with loss of tolerance to self antigens, is suggested to have apoptotic cells as the source of antigens. Foxp3, a transcription factor, is suggested to be …-
dc.languageengen_US
dc.publisherWiley-Blackwell Publishing Asia. The Journal's web site is located at http://www.blackwell-synergy.com/loi/ijrd-
dc.relation.ispartofInternational Journal of Rheumatic Diseasesen_US
dc.rightsThe definitive version is available at www3.interscience.wiley.com-
dc.subjectMedical sciences-
dc.subjectRheumatology-
dc.titleFoxp3 mRNA expression on DC subsets in patients of systemic lupus erythematosusen_US
dc.typeConference_Paperen_US
dc.identifier.emailJin, O: jinou@hkusua.hku.hken_US
dc.identifier.emailKavikondala, S: sushma@hku.hken_US
dc.identifier.emailMok, MY: temy@hkucc.hku.hken_US
dc.identifier.emailSun, LY: lingyunsun2001@yahoo.com.cnen_US
dc.identifier.emailChan, A: wkchanf@hkusua.hku.hk-
dc.identifier.emailYeung, J: joebecky@hotmail.com-
dc.identifier.emailNie, Y: luckynyj@hkusua.hku.hk-
dc.identifier.emailLau, CS: cslau@hku.hk-
dc.identifier.authorityMok, MY=rp00490en_US
dc.identifier.authorityLau, CS=rp01348en_US
dc.description.naturelink_to_OA_fulltext-
dc.identifier.doi10.1111/j.1756-185X.2010.01492.x-
dc.identifier.hkuros177413en_US
dc.identifier.volume13en_US
dc.identifier.issuesuppl. 1-
dc.identifier.spage55en_US
dc.identifier.epage55-
dc.description.otherThe 14th Congress of Asia Pacific League of Associations for Rheumatology, Hong Kong, China, 11-15 July 2010. In International Journal of Rheumatic Diseases, 2010, v. 13 suppl. 1, p. 55, abstract no. 0557-

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