Article: Schizophrenic patients and their unaffected siblings share increased resting-state connectivity in the task-negative network but not its anticorrelated task-positive network
| Title | Schizophrenic patients and their unaffected siblings share increased resting-state connectivity in the task-negative network but not its anticorrelated task-positive network | ||||||||
|---|---|---|---|---|---|---|---|---|---|
| Authors | Liu, H4 Kaneko, Y2 Ouyang, X4 Li, L4 Hao, Y4 Chen, EYH1 Jiang, T3 Zhou, Y3 Liu, Z4 | ||||||||
| Keywords | Schizophrenia Unaffected sibling Default mode network Functional connectivity Resting-state | ||||||||
| Issue Date | 2012 | ||||||||
| Publisher | Oxford University Press. The Journal's web site is located at http://schizophreniabulletin.oxfordjournals.org/ | ||||||||
| Citation | Schizophrenia Bulletin, 2012, v. 38 n. 2, p. 285-294 [How to Cite?] DOI: http://dx.doi.org/10.1093/schbul/sbq074 | ||||||||
| Abstract | Background: Abnormal connectivity of the anticorrelated intrinsic networks, the task-negative network (TNN), and the task-positive network (TPN) is implicated in schizophrenia. Comparisons between schizophrenic patients and their unaffected siblings enable further understanding of illness susceptibility and pathophysiology. We examined the resting-state connectivity differences in the intrinsic networks between schizophrenic patients, their unaffected siblings, and healthy controls. Methods: Resting-state functional magnetic resonance images were obtained from 25 individuals in each subject group. The posterior cingulate cortex/precuneus and right dorsolateral prefrontal cortex were used as seed regions to identify the TNN and TPN through functional connectivity analysis. Interregional connectivity strengths were analyzed using overlapped intrinsic networks composed of regions common to all subject groups. Results: Schizophrenic patients and their unaffected siblings showed increased connectivity in the TNN between the bilateral inferior temporal gyri. By contrast, schizophrenic patients alone demonstrated increased connectivity between the posterior cingulate cortex/precuneus and left inferior temporal gyrus and between the ventral medial prefrontal cortex and right lateral parietal cortex in the TNN. Schizophrenic patients exhibited increased connectivity between the left dorsolateral prefrontal cortex and right inferior frontal gyrus in the TPN relative to their unaffected siblings, though this trend only approached statistical significance in comparison to healthy controls. Conclusion: Resting-state hyperconnectivity of the intrinsic networks may disrupt network coordination and thereby contribute to the pathophysiology of schizophrenia. Similar, though milder, hyperconnectivity of the TNN in unaffected siblings of schizophrenic patients may contribute to the identification of schizophrenia endophenotypes and ultimately to the determination of schizophrenia risk genes. | ||||||||
| ISSN | 0586-7614 2011 Impact Factor: 8.8 2011 SCImago Journal Rankings: 0.444 | ||||||||
| DOI | http://dx.doi.org/10.1093/schbul/sbq074 | ||||||||
| ISI Accession Number ID | WOS:000300731100016
Funding Information: National Natural Science Foundation of China (30670752 to Z.L. and 30900487 to Y.Z.); National Basic Research Program of China (2006CB500808, 2007CB512300); 11th Five Year Key Program for Science and Technology Development of China (2007BAI17B05). | ||||||||
| PubMed Central ID | PMC3283150 |
| dc.contributor.author | Liu, H | ||||||||
|---|---|---|---|---|---|---|---|---|---|
| dc.contributor.author | Kaneko, Y | ||||||||
| dc.contributor.author | Ouyang, X | ||||||||
| dc.contributor.author | Li, L | ||||||||
| dc.contributor.author | Hao, Y | ||||||||
| dc.contributor.author | Chen, EYH | ||||||||
| dc.contributor.author | Jiang, T | ||||||||
| dc.contributor.author | Zhou, Y | ||||||||
| dc.contributor.author | Liu, Z | ||||||||
| dc.date.accessioned | 2010-12-23T08:35:51Z | ||||||||
| dc.date.available | 2010-12-23T08:35:51Z | ||||||||
| dc.date.issued | 2012 | ||||||||
| dc.description.abstract | Background: Abnormal connectivity of the anticorrelated intrinsic networks, the task-negative network (TNN), and the task-positive network (TPN) is implicated in schizophrenia. Comparisons between schizophrenic patients and their unaffected siblings enable further understanding of illness susceptibility and pathophysiology. We examined the resting-state connectivity differences in the intrinsic networks between schizophrenic patients, their unaffected siblings, and healthy controls. Methods: Resting-state functional magnetic resonance images were obtained from 25 individuals in each subject group. The posterior cingulate cortex/precuneus and right dorsolateral prefrontal cortex were used as seed regions to identify the TNN and TPN through functional connectivity analysis. Interregional connectivity strengths were analyzed using overlapped intrinsic networks composed of regions common to all subject groups. Results: Schizophrenic patients and their unaffected siblings showed increased connectivity in the TNN between the bilateral inferior temporal gyri. By contrast, schizophrenic patients alone demonstrated increased connectivity between the posterior cingulate cortex/precuneus and left inferior temporal gyrus and between the ventral medial prefrontal cortex and right lateral parietal cortex in the TNN. Schizophrenic patients exhibited increased connectivity between the left dorsolateral prefrontal cortex and right inferior frontal gyrus in the TPN relative to their unaffected siblings, though this trend only approached statistical significance in comparison to healthy controls. Conclusion: Resting-state hyperconnectivity of the intrinsic networks may disrupt network coordination and thereby contribute to the pathophysiology of schizophrenia. Similar, though milder, hyperconnectivity of the TNN in unaffected siblings of schizophrenic patients may contribute to the identification of schizophrenia endophenotypes and ultimately to the determination of schizophrenia risk genes. | ||||||||
| dc.description.nature | postprint | ||||||||
| dc.identifier.citation | Schizophrenia Bulletin, 2012, v. 38 n. 2, p. 285-294 [How to Cite?] DOI: http://dx.doi.org/10.1093/schbul/sbq074 | ||||||||
| dc.identifier.doi | http://dx.doi.org/10.1093/schbul/sbq074 | ||||||||
| dc.identifier.epage | 294 | ||||||||
| dc.identifier.hkuros | 188029 | ||||||||
| dc.identifier.hkuros | 177479 | ||||||||
| dc.identifier.isi | WOS:000300731100016
Funding Information: National Natural Science Foundation of China (30670752 to Z.L. and 30900487 to Y.Z.); National Basic Research Program of China (2006CB500808, 2007CB512300); 11th Five Year Key Program for Science and Technology Development of China (2007BAI17B05). | ||||||||
| dc.identifier.issn | 0586-7614 2011 Impact Factor: 8.8 2011 SCImago Journal Rankings: 0.444 | ||||||||
| dc.identifier.issue | 2 | ||||||||
| dc.identifier.pmcid | PMC3283150 | ||||||||
| dc.identifier.pmid | 20595202 | ||||||||
| dc.identifier.scopus | eid_2-s2.0-84863244364 | ||||||||
| dc.identifier.spage | 285 | ||||||||
| dc.identifier.uri | http://hdl.handle.net/10722/129353 | ||||||||
| dc.identifier.volume | 38 | ||||||||
| dc.language | eng | ||||||||
| dc.publisher | Oxford University Press. The Journal's web site is located at http://schizophreniabulletin.oxfordjournals.org/ | ||||||||
| dc.publisher.place | United Kingdom | ||||||||
| dc.relation.ispartof | Schizophrenia Bulletin | ||||||||
| dc.rights | Creative Commons: Attribution 3.0 Hong Kong License | ||||||||
| dc.rights | This is a pre-copy-editing, author-produced PDF of an article accepted for publication in Schizophrenia Bulletin following peer review. The definitive publisher-authenticated version Schizophrenia Bulletin, 2012, v. 38 n. 2, p. 285-294 is available online at: http://schizophreniabulletin.oxfordjournals.org/content/38/2/285 | ||||||||
| dc.subject | Schizophrenia | ||||||||
| dc.subject | Unaffected sibling | ||||||||
| dc.subject | Default mode network | ||||||||
| dc.subject | Functional connectivity | ||||||||
| dc.subject | Resting-state | ||||||||
| dc.title | Schizophrenic patients and their unaffected siblings share increased resting-state connectivity in the task-negative network but not its anticorrelated task-positive network | ||||||||
| dc.type | Article |
Author Affiliations
- The University of Hong Kong
- Yale University
- Chinese Academy of Sciences
- Central South University China

