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Article: Shedding dynamics of Epstein-Barr virus: A type 1 carcinogen

TitleShedding dynamics of Epstein-Barr virus: A type 1 carcinogen
Authors
KeywordsCarcinogen
Epstein-Barr virus
Shedding
Issue Date2010
PublisherPergamon. The Journal's web site is located at http://www.elsevier.com/locate/archoralbio
Citation
Archives Of Oral Biology, 2010, v. 55 n. 9, p. 639-647 How to Cite?
AbstractEpstein Barr virus (EBV) is one of the ubiquitous viral carcinogens found in humans and successfully infects more than 90% of the world population. The spectrum of EBV-related pathology ranges from asymptomatic primary infection to grave B- and T-cell malignancies. EBV triggers lymphoproliferative disorders after allogeneic stem cell transplantation, which contributes to higher mortality rates. Although the transmission of EBV primarily occurs from an infected host to a naive host through viral shedding from the oropharynx, increasing evidence points to considerable amount of shedding in other anatomical sites such as cervix, anal mucosa, breast milk and respiratory tract. It is impossible to eradicate the prevalence of EBV-related malignancies and other pathologies without preventing viral shedding. However, a detail analysis of the multifaceted nature of EBV shedding is not available in the literature. Thus, this review focuses on elucidating the key elements of the shedding dynamics of this carcinogenic virus. © 2010 Elsevier Ltd. All rights reserved.
Persistent Identifierhttp://hdl.handle.net/10722/129071
ISSN
2015 Impact Factor: 1.733
2015 SCImago Journal Rankings: 0.713
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorPerera, RAPMen_HK
dc.contributor.authorSamaranayake, LPen_HK
dc.contributor.authorTsang, CSPen_HK
dc.date.accessioned2010-12-23T08:32:04Z-
dc.date.available2010-12-23T08:32:04Z-
dc.date.issued2010en_HK
dc.identifier.citationArchives Of Oral Biology, 2010, v. 55 n. 9, p. 639-647en_HK
dc.identifier.issn0003-9969en_HK
dc.identifier.urihttp://hdl.handle.net/10722/129071-
dc.description.abstractEpstein Barr virus (EBV) is one of the ubiquitous viral carcinogens found in humans and successfully infects more than 90% of the world population. The spectrum of EBV-related pathology ranges from asymptomatic primary infection to grave B- and T-cell malignancies. EBV triggers lymphoproliferative disorders after allogeneic stem cell transplantation, which contributes to higher mortality rates. Although the transmission of EBV primarily occurs from an infected host to a naive host through viral shedding from the oropharynx, increasing evidence points to considerable amount of shedding in other anatomical sites such as cervix, anal mucosa, breast milk and respiratory tract. It is impossible to eradicate the prevalence of EBV-related malignancies and other pathologies without preventing viral shedding. However, a detail analysis of the multifaceted nature of EBV shedding is not available in the literature. Thus, this review focuses on elucidating the key elements of the shedding dynamics of this carcinogenic virus. © 2010 Elsevier Ltd. All rights reserved.en_HK
dc.languageengen_US
dc.publisherPergamon. The Journal's web site is located at http://www.elsevier.com/locate/archoralbioen_HK
dc.relation.ispartofArchives of Oral Biologyen_HK
dc.subjectCarcinogenen_HK
dc.subjectEpstein-Barr virusen_HK
dc.subjectSheddingen_HK
dc.subject.meshAnimals-
dc.subject.meshB-Lymphocytes - virology-
dc.subject.meshHerpesvirus 4, Human - physiology-
dc.subject.meshMouth Mucosa - virology-
dc.subject.meshVirus Shedding - physiology-
dc.titleShedding dynamics of Epstein-Barr virus: A type 1 carcinogenen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0003-9969&volume=55&issue=9&spage=639&epage=647&date=2010&atitle=Shedding+dynamics+of+Epstein-Barr+virus:+A+type+1+carcinogen-
dc.identifier.emailSamaranayake, LP:lakshman@hku.hken_HK
dc.identifier.emailTsang, CSP:csptsang@hkucc.hku.hken_HK
dc.identifier.authoritySamaranayake, LP=rp00023en_HK
dc.identifier.authorityTsang, CSP=rp00026en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1016/j.archoralbio.2010.06.009en_HK
dc.identifier.pmid20627195-
dc.identifier.scopuseid_2-s2.0-78149360292en_HK
dc.identifier.hkuros177038en_US
dc.identifier.hkuros217046-
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-78149360292&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume55en_HK
dc.identifier.issue9en_HK
dc.identifier.spage639en_HK
dc.identifier.epage647en_HK
dc.identifier.isiWOS:000281297500005-
dc.publisher.placeUnited Kingdomen_HK
dc.identifier.scopusauthoridPerera, RAPM=43861730800en_HK
dc.identifier.scopusauthoridSamaranayake, LP=7102761002en_HK
dc.identifier.scopusauthoridTsang, CSP=7202936002en_HK
dc.identifier.citeulike7591502-

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