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Conference Paper: Eradication of Helicobacter pylori did not accelerate the healing of gastric and duodenal ulcers / erosions in patients on long-term continuous nonsteroidal anti-inflammatory drugs

TitleEradication of Helicobacter pylori did not accelerate the healing of gastric and duodenal ulcers / erosions in patients on long-term continuous nonsteroidal anti-inflammatory drugs
Authors
Issue Date1998
PublisherWB Saunders Co. The Journal's web site is located at http://www.elsevier.com/locate/gastro
Citation
The 1998 Annual Meeting of the American Gastroenterological Association (AGA) and Digestive Disease Week, New Orleans, LA., 17–20 May 1998. In Gastroenterology, 1998, v. 114 n. suppl. 1, p. A192, abstract no. G0788 How to Cite?
AbstractINTRODUCTION: It is controversial whether Helicobacter pylori (H. pylori) acts synergistically with non-steroidal anti-inflammatory drugs (NSAIDs) to cause peptic ulcers. AIM: To study whether patients receiving NSAIDs with gastric or duodenal ulcers/erosions benefit from eradication of H. pylori infection. METHOD: 168 patients on long-term continuous NSAIDs from the Rheumatology Clinic were endoscoped during a period of 2 yrs. Group (I): 22 patients with ulcers (diameter > 3mm) and H. pylori were given ranitidine 150mg bd with or without H. pylori eradication therapy (metronidazole 300mg, amoxycillin 500mg and clarithromycin 250mg qid for 2 weeks - MAC). Ulcers healing rates were examined by 4 weekly endoscopies. Group (II): 26 patients with H. pylori and gastric or duodenal erosions were randomized to receive either (1) 2 weeks MAC qid or (2) 2 weeks placebo qid. Endoscopy was performed at the end of 12 wks with complete healing defined as no erosions, partial healing as reduced number of erosions and unhealing as same number of erosions detected. NSAIDs were continued in all patients. RESULTS: No patients suffered from clinical gastrointestinal bleeding at the end of study …
Persistent Identifierhttp://hdl.handle.net/10722/102926
ISSN
2015 Impact Factor: 18.187
2015 SCImago Journal Rankings: 7.170

 

DC FieldValueLanguage
dc.contributor.authorLai, KC-
dc.contributor.authorLam, SK-
dc.contributor.authorHui, WM-
dc.contributor.authorWong, BCY-
dc.contributor.authorLau, WCS-
dc.contributor.authorHu, HC-
dc.contributor.authorWen, ZF-
dc.date.accessioned2010-09-25T20:50:28Z-
dc.date.available2010-09-25T20:50:28Z-
dc.date.issued1998-
dc.identifier.citationThe 1998 Annual Meeting of the American Gastroenterological Association (AGA) and Digestive Disease Week, New Orleans, LA., 17–20 May 1998. In Gastroenterology, 1998, v. 114 n. suppl. 1, p. A192, abstract no. G0788-
dc.identifier.issn0016-5085-
dc.identifier.urihttp://hdl.handle.net/10722/102926-
dc.description.abstractINTRODUCTION: It is controversial whether Helicobacter pylori (H. pylori) acts synergistically with non-steroidal anti-inflammatory drugs (NSAIDs) to cause peptic ulcers. AIM: To study whether patients receiving NSAIDs with gastric or duodenal ulcers/erosions benefit from eradication of H. pylori infection. METHOD: 168 patients on long-term continuous NSAIDs from the Rheumatology Clinic were endoscoped during a period of 2 yrs. Group (I): 22 patients with ulcers (diameter > 3mm) and H. pylori were given ranitidine 150mg bd with or without H. pylori eradication therapy (metronidazole 300mg, amoxycillin 500mg and clarithromycin 250mg qid for 2 weeks - MAC). Ulcers healing rates were examined by 4 weekly endoscopies. Group (II): 26 patients with H. pylori and gastric or duodenal erosions were randomized to receive either (1) 2 weeks MAC qid or (2) 2 weeks placebo qid. Endoscopy was performed at the end of 12 wks with complete healing defined as no erosions, partial healing as reduced number of erosions and unhealing as same number of erosions detected. NSAIDs were continued in all patients. RESULTS: No patients suffered from clinical gastrointestinal bleeding at the end of study …-
dc.languageeng-
dc.publisherWB Saunders Co. The Journal's web site is located at http://www.elsevier.com/locate/gastro-
dc.relation.ispartofGastroenterology-
dc.rightsPosting accepted manuscript (postprint): © <year>. This manuscript version is made available under the CC-BY-NC-ND 4.0 license http://creativecommons.org/licenses/by-nc-nd/4.0/-
dc.titleEradication of Helicobacter pylori did not accelerate the healing of gastric and duodenal ulcers / erosions in patients on long-term continuous nonsteroidal anti-inflammatory drugs-
dc.typeConference_Paper-
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0016-5085&volume=114&issue=4&spage=A192&epage=&date=1998&atitle=Eradication+of+Helicobacter+pylori+did+not+accelerate+the+healing+of+gastric+and+duodenal+ulcers+/+erosions+in+patients+on+long-term+continuous+nonsteroidal+anti-inflammatory+drugsen_HK
dc.identifier.emailLai, KC: kclai@hku.hk-
dc.identifier.emailLam, SK: hrmelsk@hkucc.hku.hk-
dc.identifier.emailHui, WM: hrmehwm@hkucc.hku.hk-
dc.identifier.emailWong, BCY: bcywong@hku.hk-
dc.identifier.emailLau, WCS: cslau@hku.hk-
dc.identifier.emailHu, HC: hchu@hkucc.hku.hk-
dc.identifier.authorityWong, BCY=rp00429-
dc.identifier.authorityLau, WCS=rp01348-
dc.description.natureLink_to_subscribed_fulltext-
dc.identifier.doi10.1016/S0016-5085(98)80782-8-
dc.identifier.hkuros31638-
dc.identifier.volume114-
dc.identifier.issuesuppl. 1-
dc.identifier.spageA192, abstract no. G0788-
dc.identifier.epageA192, abstract no. G0788-
dc.publisher.placeUnited States-

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