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Conference Paper: Overproduction of nitric oxide from neuronal nitric oxide synthase during ischemia is partly mediated via activation of neuropeptide Y Y1 receptors

TitleOverproduction of nitric oxide from neuronal nitric oxide synthase during ischemia is partly mediated via activation of neuropeptide Y Y1 receptors
Authors
Issue Date2004
PublisherLippincott Williams & Wilkins. The Journal's web site is located at http://www.neurology.org
Citation
Neurology, v. 62 n. Suppl 5, p. A144 How to Cite?
Persistent Identifierhttp://hdl.handle.net/10722/102110
ISSN
2015 Impact Factor: 8.166
2015 SCImago Journal Rankings: 3.691

 

DC FieldValueLanguage
dc.contributor.authorChan, PSen_HK
dc.contributor.authorShen, Jen_HK
dc.contributor.authorFung, PCWen_HK
dc.contributor.authorCheung, RTFen_HK
dc.date.accessioned2010-09-25T20:17:27Z-
dc.date.available2010-09-25T20:17:27Z-
dc.date.issued2004en_HK
dc.identifier.citationNeurology, v. 62 n. Suppl 5, p. A144en_HK
dc.identifier.issn0028-3878en_HK
dc.identifier.urihttp://hdl.handle.net/10722/102110-
dc.languageengen_HK
dc.publisherLippincott Williams & Wilkins. The Journal's web site is located at http://www.neurology.orgen_HK
dc.relation.ispartofNeurologyen_HK
dc.rightsNeurology. Copyright © Lippincott Williams & Wilkins.en_HK
dc.titleOverproduction of nitric oxide from neuronal nitric oxide synthase during ischemia is partly mediated via activation of neuropeptide Y Y1 receptorsen_HK
dc.typeConference_Paperen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0028-3878&volume=62&issue=Suppl 5&spage=A144&epage=&date=2004&atitle=Overproduction+of+nitric+oxide+from+neuronal+nitric+oxide+synthase+during+ischemia+is+partly+mediated+via+activation+of+neuropeptide+Y+Y1+receptorsen_HK
dc.identifier.emailShen, J: shenjg@hkucc.hku.hken_HK
dc.identifier.emailFung, PCW: hrspfcw@hku.hken_HK
dc.identifier.emailCheung, RTF: rtcheung@hku.hken_HK
dc.identifier.authorityShen, J=rp00487en_HK
dc.identifier.authorityCheung, RTF=rp00434en_HK
dc.identifier.hkuros87593en_HK
dc.identifier.volume, v. 62 n. Suppl 5en_HK
dc.identifier.issueSuppl 5en_HK
dc.identifier.spage144en_HK

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