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Article: Gap junction inhibition by heptanol increases ventricular arrhythmogenicity by decreasing conduction velocity without affecting repolarization properties or myocardial refractoriness in Langendorff-perfused mouse hearts

TitleGap junction inhibition by heptanol increases ventricular arrhythmogenicity by decreasing conduction velocity without affecting repolarization properties or myocardial refractoriness in Langendorff-perfused mouse hearts
Authors
Issue Date2016
Citation
Molecular Medicine Reports (Forthcoming) How to Cite?
Persistent Identifierhttp://hdl.handle.net/10722/227127

 

DC FieldValueLanguage
dc.contributor.authorTse, G-
dc.contributor.authorYeo, JM-
dc.contributor.authorTse, V-
dc.contributor.authorSun, B-
dc.date.accessioned2016-07-18T09:08:36Z-
dc.date.available2016-07-18T09:08:36Z-
dc.date.issued2016-
dc.identifier.citationMolecular Medicine Reports (Forthcoming)-
dc.identifier.urihttp://hdl.handle.net/10722/227127-
dc.languageeng-
dc.relation.ispartofMolecular Medicine Reports-
dc.titleGap junction inhibition by heptanol increases ventricular arrhythmogenicity by decreasing conduction velocity without affecting repolarization properties or myocardial refractoriness in Langendorff-perfused mouse hearts-
dc.typeArticle-
dc.identifier.emailTse, G: tseg@hku.hk-
dc.identifier.authorityTse, G=rp02073-
dc.identifier.hkuros259501-

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