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Conference Paper: Impaired cold tolerance in EP4 knockout mice is not due to compromised recruitment of UCP1
| Title | Impaired cold tolerance in EP4 knockout mice is not due to compromised recruitment of UCP1 |
|---|---|
| Authors | |
| Issue Date | 2014 |
| Publisher | Medcom Limited. The Journal's web site is located at http://www.hkcchk.com/journals.php#3 |
| Citation | The 18th Annual Scientific Meeting of the Institute of Cardiovascular Science and Medicine (ICSM 2014), Hong Kong, 1 November 2014. In Journal of the Hong Kong College of Cardiology, 2014, v. 22 n. 2, p. 56, abstract P02 How to Cite? |
| Abstract | OBJECTIVE: Uncoupling protein 1 (UCP1) is the key component of beta-adrenergically controlled non-shivering thermogenesis in brown adipose tissue (BAT). Certain white adipose tissue (WAT), upon exposition to cold, can undergo a process knowing as browning where it takes on characteristics of BAT, notably induction of UCP1 and the presence of multilocular lipid droplets and multiple mitochondria. Our preliminary data indicated that mice deficient in EP4 (one subtype of PGE2 receptor) are cold-intolerant, suggesting that EP4 influence thermogenesis capacity. The aim of this project was to investigate whether or not the impaired thermogenesis in ... |
| Description | Abstracts for Posters: no. P02 This journal issue including abstracts of 18th Annual Scientific Meeting Institute of Cardiovascular Science and Medicine |
| Persistent Identifier | http://hdl.handle.net/10722/217608 |
| ISSN | 2020 SCImago Journal Rankings: 0.105 |
| DC Field | Value | Language |
|---|---|---|
| dc.contributor.author | YING, F | - |
| dc.contributor.author | Cai, Y | - |
| dc.contributor.author | Tang, EHC | - |
| dc.date.accessioned | 2015-09-18T06:07:03Z | - |
| dc.date.available | 2015-09-18T06:07:03Z | - |
| dc.date.issued | 2014 | - |
| dc.identifier.citation | The 18th Annual Scientific Meeting of the Institute of Cardiovascular Science and Medicine (ICSM 2014), Hong Kong, 1 November 2014. In Journal of the Hong Kong College of Cardiology, 2014, v. 22 n. 2, p. 56, abstract P02 | - |
| dc.identifier.issn | 1027-7811 | - |
| dc.identifier.uri | http://hdl.handle.net/10722/217608 | - |
| dc.description | Abstracts for Posters: no. P02 | - |
| dc.description | This journal issue including abstracts of 18th Annual Scientific Meeting Institute of Cardiovascular Science and Medicine | - |
| dc.description.abstract | OBJECTIVE: Uncoupling protein 1 (UCP1) is the key component of beta-adrenergically controlled non-shivering thermogenesis in brown adipose tissue (BAT). Certain white adipose tissue (WAT), upon exposition to cold, can undergo a process knowing as browning where it takes on characteristics of BAT, notably induction of UCP1 and the presence of multilocular lipid droplets and multiple mitochondria. Our preliminary data indicated that mice deficient in EP4 (one subtype of PGE2 receptor) are cold-intolerant, suggesting that EP4 influence thermogenesis capacity. The aim of this project was to investigate whether or not the impaired thermogenesis in ... | - |
| dc.language | eng | - |
| dc.publisher | Medcom Limited. The Journal's web site is located at http://www.hkcchk.com/journals.php#3 | - |
| dc.relation.ispartof | Journal of the Hong Kong College of Cardiology | - |
| dc.title | Impaired cold tolerance in EP4 knockout mice is not due to compromised recruitment of UCP1 | - |
| dc.type | Conference_Paper | - |
| dc.identifier.email | Cai, Y: caidavid@HKUCC-COM.hku.hk | - |
| dc.identifier.email | Tang, EHC: evatang1@hku.hk | - |
| dc.identifier.authority | Tang, EHC=rp01382 | - |
| dc.description.nature | postprint | - |
| dc.identifier.hkuros | 250427 | - |
| dc.identifier.volume | 22 | - |
| dc.identifier.issue | 2 | - |
| dc.identifier.spage | 56, abstract P02 | - |
| dc.identifier.epage | 56, abstract P02 | - |
| dc.publisher.place | Hong Kong | - |
| dc.identifier.issnl | 1027-7811 | - |