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Article: Immune dysregulation in minimal change nephropathy

TitleImmune dysregulation in minimal change nephropathy
Authors
Mathieson, PW
KeywordsImmune dysregulation
Minimal change disease
Nephrotic syndrome
Issue Date2003
Citation
Nephrology Dialysis Transplantation, 2003, v. 18 n. SUPPL. 6, p. vi26-vi29 How to Cite?
AbstractMinimal change nephropathy (MCN) has been associated with a wide variety of abnormalities in the immune response of affected individuals. Many of these may be a consequence of the renal lesion, rather than a cause thereof. This article reviews some of these immunological phenomena and concentrates in particular on the imbalance between type 1 and type 2 cytokine responses, suggesting that there are plausible mechanisms whereby a type 2 cytokine bias could directly or indirectly cause MCN.
Persistent Identifierhttp://hdl.handle.net/10722/195385
ISSN
0931-0509
2021 Impact Factor: 7.186
2020 SCImago Journal Rankings: 1.654

 

DC FieldValueLanguage
dc.contributor.authorMathieson, PW-
dc.date.accessioned2014-02-28T06:12:04Z-
dc.date.available2014-02-28T06:12:04Z-
dc.date.issued2003-
dc.identifier.citationNephrology Dialysis Transplantation, 2003, v. 18 n. SUPPL. 6, p. vi26-vi29-
dc.identifier.issn0931-0509-
dc.identifier.urihttp://hdl.handle.net/10722/195385-
dc.description.abstractMinimal change nephropathy (MCN) has been associated with a wide variety of abnormalities in the immune response of affected individuals. Many of these may be a consequence of the renal lesion, rather than a cause thereof. This article reviews some of these immunological phenomena and concentrates in particular on the imbalance between type 1 and type 2 cytokine responses, suggesting that there are plausible mechanisms whereby a type 2 cytokine bias could directly or indirectly cause MCN.-
dc.languageeng-
dc.relation.ispartofNephrology Dialysis Transplantation-
dc.subjectImmune dysregulation-
dc.subjectMinimal change disease-
dc.subjectNephrotic syndrome-
dc.titleImmune dysregulation in minimal change nephropathy-
dc.typeArticle-
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.pmid12953038-
dc.identifier.scopuseid_2-s2.0-0041829301-
dc.identifier.volume18-
dc.identifier.issueSUPPL. 6-
dc.identifier.spagevi26-
dc.identifier.epagevi29-
dc.identifier.issnl0931-0509-

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