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Conference Paper: Molecular aspects of left ventricular hypertrophy / Hypertrophie Ventriculaire Gauche: Aspects Moleculaires

TitleMolecular aspects of left ventricular hypertrophy / Hypertrophie Ventriculaire Gauche: Aspects Moleculaires
Authors
Issue Date1993
Citation
Archives des Maladies du Coeur et des Vaisseaux, 1993, v. 86 Spec. Iss. 1, p. 73-75 How to Cite?
AbstractThe expression of genes changes during left ventricular hypertrophy. These changes are known as mechanogenic transduction. With respect to the two main contractile proteins, myosin and actin, this is due to a differential expression of their respective multigenic families. Changes of isoform are uncoordinated in time and space. It is important to recognise that the genes themselves are normal and that it is their regulation which is disturbed. Recent data shows that, under normal conditions, the regulation occurs at the transcription stage. A full understanding of these mechanisms is necessary if we are to comprehend the pathogenesis of cardiac hypertrophy and failure.
Persistent Identifierhttp://hdl.handle.net/10722/195232
ISSN
2009 Impact Factor: 0.398
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorSchwartz, K-
dc.contributor.authorChassagne, C-
dc.contributor.authorCarrier, L-
dc.contributor.authorBoheler, KR-
dc.date.accessioned2014-02-25T01:40:20Z-
dc.date.available2014-02-25T01:40:20Z-
dc.date.issued1993-
dc.identifier.citationArchives des Maladies du Coeur et des Vaisseaux, 1993, v. 86 Spec. Iss. 1, p. 73-75-
dc.identifier.issn0003-9683-
dc.identifier.urihttp://hdl.handle.net/10722/195232-
dc.description.abstractThe expression of genes changes during left ventricular hypertrophy. These changes are known as mechanogenic transduction. With respect to the two main contractile proteins, myosin and actin, this is due to a differential expression of their respective multigenic families. Changes of isoform are uncoordinated in time and space. It is important to recognise that the genes themselves are normal and that it is their regulation which is disturbed. Recent data shows that, under normal conditions, the regulation occurs at the transcription stage. A full understanding of these mechanisms is necessary if we are to comprehend the pathogenesis of cardiac hypertrophy and failure.-
dc.languageeng-
dc.relation.ispartofArchives des Maladies du Coeur et des Vaisseaux-
dc.titleMolecular aspects of left ventricular hypertrophy / Hypertrophie Ventriculaire Gauche: Aspects Moleculaires-
dc.typeConference_Paper-
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.pmid8215783-
dc.identifier.scopuseid_2-s2.0-0027343534-
dc.identifier.volume86-
dc.identifier.issueSpec. Iss. 1-
dc.identifier.spage73-
dc.identifier.epage75-
dc.identifier.isiWOS:A1993KK57000009-
dc.customcontrol.immutablesml 160520 amended-

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