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Article: Diabetic retinopathy and systemic vascular complications

TitleDiabetic retinopathy and systemic vascular complications
Authors
Issue Date2008
PublisherPergamon. The Journal's web site is located at http://www.elsevier.com/locate/preteyeres
Citation
Progress In Retinal And Eye Research, 2008, v. 27 n. 2, p. 161-176 How to Cite?
AbstractRetinopathy is the most common complication of diabetes. The assessment of retinopathy signs presents clinicians a unique opportunity to directly visualize and assess the actual morphology of diabetic microvascular damage. Extensive studies have now shown that people with diabetic retinopathy have excess risks of systemic vascular complications, including subclinical and clinical stroke, coronary heart disease, heart failure, and nephropathy. There is also emerging evidence to suggest that diabetic retinopathy may share common genetic linkages with systemic vascular complications. The extant literature, therefore, supports the theory that diabetic retinopathy reflects widespread microcirculatory disease not only in the eye but also vital organs elsewhere in the body. Being a uniquely specific and non-invasively assessable measure of diabetic microvascular damage, retinopathy may also be envisioned as a novel biomarker of vascular disease risk in asymptomatic patients with diabetes. This review summarizes recent studies on the systemic associations of diabetic retinopathy, and discusses their pathophysiological significance and clinical implications. © 2007 Elsevier Ltd. All rights reserved.
Persistent Identifierhttp://hdl.handle.net/10722/183539
ISSN
2015 Impact Factor: 9.394
2015 SCImago Journal Rankings: 5.073
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorCheung, Nen_US
dc.contributor.authorWong, TYen_US
dc.date.accessioned2013-05-28T06:14:32Z-
dc.date.available2013-05-28T06:14:32Z-
dc.date.issued2008en_US
dc.identifier.citationProgress In Retinal And Eye Research, 2008, v. 27 n. 2, p. 161-176en_US
dc.identifier.issn1350-9462en_US
dc.identifier.urihttp://hdl.handle.net/10722/183539-
dc.description.abstractRetinopathy is the most common complication of diabetes. The assessment of retinopathy signs presents clinicians a unique opportunity to directly visualize and assess the actual morphology of diabetic microvascular damage. Extensive studies have now shown that people with diabetic retinopathy have excess risks of systemic vascular complications, including subclinical and clinical stroke, coronary heart disease, heart failure, and nephropathy. There is also emerging evidence to suggest that diabetic retinopathy may share common genetic linkages with systemic vascular complications. The extant literature, therefore, supports the theory that diabetic retinopathy reflects widespread microcirculatory disease not only in the eye but also vital organs elsewhere in the body. Being a uniquely specific and non-invasively assessable measure of diabetic microvascular damage, retinopathy may also be envisioned as a novel biomarker of vascular disease risk in asymptomatic patients with diabetes. This review summarizes recent studies on the systemic associations of diabetic retinopathy, and discusses their pathophysiological significance and clinical implications. © 2007 Elsevier Ltd. All rights reserved.en_US
dc.languageengen_US
dc.publisherPergamon. The Journal's web site is located at http://www.elsevier.com/locate/preteyeresen_US
dc.relation.ispartofProgress in Retinal and Eye Researchen_US
dc.subject.meshAnimalsen_US
dc.subject.meshDiabetic Retinopathy - Complications - Genetics - Physiopathologyen_US
dc.subject.meshHumansen_US
dc.subject.meshVascular Diseases - Complications - Genetics - Physiopathologyen_US
dc.titleDiabetic retinopathy and systemic vascular complicationsen_US
dc.typeArticleen_US
dc.identifier.emailCheung, N: dannycheung@hotmail.comen_US
dc.identifier.authorityCheung, N=rp01752en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1016/j.preteyeres.2007.12.001en_US
dc.identifier.pmid18249026-
dc.identifier.scopuseid_2-s2.0-40249097578en_US
dc.identifier.volume27en_US
dc.identifier.issue2en_US
dc.identifier.spage161en_US
dc.identifier.epage176en_US
dc.identifier.isiWOS:000255104200002-
dc.publisher.placeUnited Kingdomen_US
dc.identifier.scopusauthoridCheung, N=8054683900en_US
dc.identifier.scopusauthoridWong, TY=7403531208en_US

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