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Article: Markers of oxidative damage are not elevated in otherwise healthy individuals with the metabolic syndrome

TitleMarkers of oxidative damage are not elevated in otherwise healthy individuals with the metabolic syndrome
Authors
Issue Date2010
PublisherAmerican Diabetes Association. The Journal's web site is located at http://diabetes.diabetesjournals.org/
Citation
Diabetes Care, 2010, v. 33 n. 5, p. 1140-1142 How to Cite?
AbstractOBJECTIVE- The role of oxidative damage in the pathogenesis of metabolic syndrome is poorly understood. RESEARCH DESIGN AND METHODS- A detailed cross-sectional study was performed to assess the relationship between lipid oxidation products, γ-glutamyltransferase, highsensitivity C-reactive protein (hs-CRP), and phospholipase activities with respect to the metabolic status in a cohort of otherwise healthy individuals. RESULTS- A total of 179 individuals (87 men and 92 women) aged 43 ± 14 years (mean ± SD) participated in this study. There were no differences in the levels of plasma F 2-isoprostanes, hydroxyeicosatetraenoic acids, cholesterol oxidation products, and phospholipase activities in individuals with features of metabolic syndrome. In multivariate analyses, serum hs-CRP was a consistent independent predictor of metabolic syndrome. CONCLUSIONS- Minimal changes were observed in multiple markers of oxidative damage in a well-characterized cohort of individuals with features of metabolic syndrome. © 2010 by the American Diabetes Association.
Persistent Identifierhttp://hdl.handle.net/10722/179203
ISSN
2015 Impact Factor: 8.934
2015 SCImago Journal Rankings: 5.827
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorSeet, RCSen_US
dc.contributor.authorLee, CYJen_US
dc.contributor.authorLim, ECHen_US
dc.contributor.authorQuek, AMLen_US
dc.contributor.authorHuang, SHen_US
dc.contributor.authorKhoo, CMen_US
dc.contributor.authorHalliwell, Ben_US
dc.date.accessioned2012-12-19T09:52:54Z-
dc.date.available2012-12-19T09:52:54Z-
dc.date.issued2010en_US
dc.identifier.citationDiabetes Care, 2010, v. 33 n. 5, p. 1140-1142en_US
dc.identifier.issn0149-5992en_US
dc.identifier.urihttp://hdl.handle.net/10722/179203-
dc.description.abstractOBJECTIVE- The role of oxidative damage in the pathogenesis of metabolic syndrome is poorly understood. RESEARCH DESIGN AND METHODS- A detailed cross-sectional study was performed to assess the relationship between lipid oxidation products, γ-glutamyltransferase, highsensitivity C-reactive protein (hs-CRP), and phospholipase activities with respect to the metabolic status in a cohort of otherwise healthy individuals. RESULTS- A total of 179 individuals (87 men and 92 women) aged 43 ± 14 years (mean ± SD) participated in this study. There were no differences in the levels of plasma F 2-isoprostanes, hydroxyeicosatetraenoic acids, cholesterol oxidation products, and phospholipase activities in individuals with features of metabolic syndrome. In multivariate analyses, serum hs-CRP was a consistent independent predictor of metabolic syndrome. CONCLUSIONS- Minimal changes were observed in multiple markers of oxidative damage in a well-characterized cohort of individuals with features of metabolic syndrome. © 2010 by the American Diabetes Association.en_US
dc.languageengen_US
dc.publisherAmerican Diabetes Association. The Journal's web site is located at http://diabetes.diabetesjournals.org/en_US
dc.relation.ispartofDiabetes Careen_US
dc.titleMarkers of oxidative damage are not elevated in otherwise healthy individuals with the metabolic syndromeen_US
dc.typeArticleen_US
dc.identifier.emailLee, CYJ: jettylee@hku.hken_US
dc.identifier.authorityLee, CYJ=rp01511en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.2337/dc09-2124en_US
dc.identifier.pmid20185735-
dc.identifier.scopuseid_2-s2.0-77954921980en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-77954921980&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume33en_US
dc.identifier.issue5en_US
dc.identifier.spage1140en_US
dc.identifier.epage1142en_US
dc.identifier.eissn1935-5548-
dc.identifier.isiWOS:000277631200038-
dc.publisher.placeUnited Statesen_US
dc.identifier.scopusauthoridSeet, RCS=10045357300en_US
dc.identifier.scopusauthoridLee, CYJ=13104265200en_US
dc.identifier.scopusauthoridLim, ECH=8945547100en_US
dc.identifier.scopusauthoridQuek, AML=13605538000en_US
dc.identifier.scopusauthoridHuang, SH=8367750600en_US
dc.identifier.scopusauthoridKhoo, CM=15842051800en_US
dc.identifier.scopusauthoridHalliwell, B=7101878919en_US

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