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Article: Say NO to ET

TitleSay NO to ET
Authors
Issue Date2000
PublisherElsevier BV. The Journal's web site is located at http://www.elsevier.com/locate/autneu
Citation
Journal Of The Autonomic Nervous System, 2000, v. 81 n. 1-3, p. 271-277 How to Cite?
AbstractThe endothelial cells relase both relaxing [nitric oxide (NO), endothelium-derived hyperpolarizing factor (EDHF), prostacyclin] and contracting factors [endoperoxides, thromboxane A2, superoxide anions, endothelin-1 (ET)]. The production of ET is inhibited by NO. The latter also strongly opposes the direct effects of the former on vascular smooth muscle. With aging and vascular disease, the production of enothelial NO declines, and thus ET can be released, act and contribute to the symptoms. (C) 2000 Elsevier Science B.V.
Persistent Identifierhttp://hdl.handle.net/10722/171251
ISSN
2002 Impact Factor: 2.895
ISI Accession Number ID
References

 

DC FieldValueLanguage
dc.contributor.authorVanhoutte, PMen_US
dc.date.accessioned2012-10-30T06:12:57Z-
dc.date.available2012-10-30T06:12:57Z-
dc.date.issued2000en_US
dc.identifier.citationJournal Of The Autonomic Nervous System, 2000, v. 81 n. 1-3, p. 271-277en_US
dc.identifier.issn0165-1838en_US
dc.identifier.urihttp://hdl.handle.net/10722/171251-
dc.description.abstractThe endothelial cells relase both relaxing [nitric oxide (NO), endothelium-derived hyperpolarizing factor (EDHF), prostacyclin] and contracting factors [endoperoxides, thromboxane A2, superoxide anions, endothelin-1 (ET)]. The production of ET is inhibited by NO. The latter also strongly opposes the direct effects of the former on vascular smooth muscle. With aging and vascular disease, the production of enothelial NO declines, and thus ET can be released, act and contribute to the symptoms. (C) 2000 Elsevier Science B.V.en_US
dc.languageengen_US
dc.publisherElsevier BV. The Journal's web site is located at http://www.elsevier.com/locate/autneuen_US
dc.relation.ispartofJournal of the Autonomic Nervous Systemen_US
dc.subject.meshAnimalsen_US
dc.subject.meshEndothelin-1 - Physiologyen_US
dc.subject.meshEndothelium, Vascular - Physiologyen_US
dc.subject.meshHumansen_US
dc.subject.meshNitric Oxide - Physiologyen_US
dc.subject.meshVascular Diseases - Physiopathologyen_US
dc.titleSay NO to ETen_US
dc.typeArticleen_US
dc.identifier.emailVanhoutte, PM:vanhoutt@hku.hken_US
dc.identifier.authorityVanhoutte, PM=rp00238en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1016/S0165-1838(00)00126-0en_US
dc.identifier.pmid10869731-
dc.identifier.scopuseid_2-s2.0-0034601277en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0034601277&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume81en_US
dc.identifier.issue1-3en_US
dc.identifier.spage271en_US
dc.identifier.epage277en_US
dc.identifier.isiWOS:000088232000040-
dc.publisher.placeNetherlandsen_US
dc.identifier.scopusauthoridVanhoutte, PM=7202304247en_US
dc.identifier.citeulike10444523-

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