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Article: Regulation of adhesion molecules: A new target for the treatment of chronic venous insufficiency

TitleRegulation of adhesion molecules: A new target for the treatment of chronic venous insufficiency
Authors
KeywordsAdhesion Molecules
Chronic Venous Insufficiency
Microcirculation
S 17834
Issue Date2000
Citation
Microcirculation, 2000, v. 7 SUPPL.1, p. S41-S48 How to Cite?
AbstractAs more insight into the mechanisms leading to chronic venous insufficiency (CVI) is gained, novel targets for drug treatment of the disease, or of its complications, become available. Studies using chemical entities capable of inhibiting leukocyte adhesion in postcapillary venules have led to the discovery of selective inhibitors of cell adhesion mechanisms. The aim of the current review is to describe the pharmacology, biochemistry, and molecular biology studies performed with some new inhibitors of adhesion molecule expression. Compounds such as hydroxy triallyl farnisine (S 17834) may offer new and efficient treatment of the microcirculatory complications that accompany chronic venous disease. © 2000 Informa UK Ltd All rights reserved.
Persistent Identifierhttp://hdl.handle.net/10722/171250
ISSN
2015 Impact Factor: 2.435
2015 SCImago Journal Rankings: 1.325
References

 

DC FieldValueLanguage
dc.contributor.authorVerbeuren, TJen_US
dc.contributor.authorBouskela, Een_US
dc.contributor.authorCohen, RAen_US
dc.contributor.authorVanhoutte, PMen_US
dc.date.accessioned2012-10-30T06:12:57Z-
dc.date.available2012-10-30T06:12:57Z-
dc.date.issued2000en_US
dc.identifier.citationMicrocirculation, 2000, v. 7 SUPPL.1, p. S41-S48en_US
dc.identifier.issn1073-9688en_US
dc.identifier.urihttp://hdl.handle.net/10722/171250-
dc.description.abstractAs more insight into the mechanisms leading to chronic venous insufficiency (CVI) is gained, novel targets for drug treatment of the disease, or of its complications, become available. Studies using chemical entities capable of inhibiting leukocyte adhesion in postcapillary venules have led to the discovery of selective inhibitors of cell adhesion mechanisms. The aim of the current review is to describe the pharmacology, biochemistry, and molecular biology studies performed with some new inhibitors of adhesion molecule expression. Compounds such as hydroxy triallyl farnisine (S 17834) may offer new and efficient treatment of the microcirculatory complications that accompany chronic venous disease. © 2000 Informa UK Ltd All rights reserved.en_US
dc.languageengen_US
dc.relation.ispartofMicrocirculationen_US
dc.subjectAdhesion Moleculesen_US
dc.subjectChronic Venous Insufficiencyen_US
dc.subjectMicrocirculationen_US
dc.subjectS 17834en_US
dc.titleRegulation of adhesion molecules: A new target for the treatment of chronic venous insufficiencyen_US
dc.typeArticleen_US
dc.identifier.emailVanhoutte, PM:vanhoutt@hku.hken_US
dc.identifier.authorityVanhoutte, PM=rp00238en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1080/mic.7.S1.S41.S48en_US
dc.identifier.scopuseid_2-s2.0-0034565770en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0034565770&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume7en_US
dc.identifier.issueSUPPL.1en_US
dc.identifier.spageS41en_US
dc.identifier.epageS48en_US
dc.identifier.scopusauthoridVerbeuren, TJ=7007006534en_US
dc.identifier.scopusauthoridBouskela, E=7006976211en_US
dc.identifier.scopusauthoridCohen, RA=35562815800en_US
dc.identifier.scopusauthoridVanhoutte, PM=7202304247en_US

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