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Article: Haemorrhagic shock encephalopathy

TitleHaemorrhagic shock encephalopathy
Authors
Issue Date1989
Citation
Acta Paediatrica Scandinavica, 1989, v. 78 n. 3, p. 458-462 How to Cite?
AbstractThree children suffered from acute onset of convulsions and progressive coma associated with hyperpyrexia, profound shock and generalized bleeding tendency. No causative agent could be identified. Despite aggressive resuscitation they all died. Post-mortem examination revealed cerebral oedema, petechial haemorrhages of the gut, lungs and kidneys, and a generalized depletion of lymphocytes in the lymphoid organs. Features were compatible with haemorrhagic shock encephalopathy, which is a highly fatal disease. The possible role of hyperpyrexia in its pathophysiology is discussed. More careful case identification and research in various possible aetiological factors may help elucidate its pathogenesis.
Persistent Identifierhttp://hdl.handle.net/10722/170239
ISSN
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorTam, AYCen_US
dc.contributor.authorNg, IOLen_US
dc.contributor.authorCheung, PTen_US
dc.contributor.authorTang, TSen_US
dc.contributor.authorLi, CHen_US
dc.date.accessioned2012-10-30T06:06:54Z-
dc.date.available2012-10-30T06:06:54Z-
dc.date.issued1989en_US
dc.identifier.citationActa Paediatrica Scandinavica, 1989, v. 78 n. 3, p. 458-462en_US
dc.identifier.issn0001-656Xen_US
dc.identifier.urihttp://hdl.handle.net/10722/170239-
dc.description.abstractThree children suffered from acute onset of convulsions and progressive coma associated with hyperpyrexia, profound shock and generalized bleeding tendency. No causative agent could be identified. Despite aggressive resuscitation they all died. Post-mortem examination revealed cerebral oedema, petechial haemorrhages of the gut, lungs and kidneys, and a generalized depletion of lymphocytes in the lymphoid organs. Features were compatible with haemorrhagic shock encephalopathy, which is a highly fatal disease. The possible role of hyperpyrexia in its pathophysiology is discussed. More careful case identification and research in various possible aetiological factors may help elucidate its pathogenesis.en_US
dc.languageengen_US
dc.relation.ispartofActa Paediatrica Scandinavicaen_US
dc.subject.meshBrain - Pathologyen_US
dc.subject.meshBrain Diseases - Etiology - Physiopathologyen_US
dc.subject.meshChild, Preschoolen_US
dc.subject.meshComa - Physiopathologyen_US
dc.subject.meshFemaleen_US
dc.subject.meshFever - Physiopathologyen_US
dc.subject.meshHeat Exhaustionen_US
dc.subject.meshHumansen_US
dc.subject.meshInfanten_US
dc.subject.meshMaleen_US
dc.subject.meshNecrosisen_US
dc.subject.meshShock, Hemorrhagic - Physiopathologyen_US
dc.subject.meshSyndromeen_US
dc.titleHaemorrhagic shock encephalopathyen_US
dc.typeArticleen_US
dc.identifier.emailNg, IOL:iolng@hkucc.hku.hken_US
dc.identifier.emailCheung, PT:ptcheung@hkucc.hku.hken_US
dc.identifier.authorityNg, IOL=rp00335en_US
dc.identifier.authorityCheung, PT=rp00351en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1111/j.1651-2227.1989.tb11111.x-
dc.identifier.pmid2741690-
dc.identifier.scopuseid_2-s2.0-0024547760en_US
dc.identifier.volume78en_US
dc.identifier.issue3en_US
dc.identifier.spage458en_US
dc.identifier.epage462en_US
dc.identifier.isiWOS:A1989U759000024-
dc.identifier.scopusauthoridTam, AYC=7101744914en_US
dc.identifier.scopusauthoridNg, IOL=7102753722en_US
dc.identifier.scopusauthoridCheung, PT=7202595465en_US
dc.identifier.scopusauthoridTang, TS=7401988573en_US
dc.identifier.scopusauthoridLi, CH=7501679305en_US

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