Dr Ballard, Heather Janet
Dr. Ballard obtained her BSc and PhD from the University of Leeds. Upon graduation she spent 3 years in a junior Faculty position at the University of Newcastle upon Tyne, followed by a brief spell in the pharmaceutical industry, before joining the University of Hong Kong as a Lecturer in 1988. She was promoted to Senior Lecturer/Associate Professor in 1997.
Dr. Ballard is committed to excellence in both teaching and research: she served the Faculty as Assistant Dean for Education & Student Affairs from 2004-2010 and is currently the Deputy Director of the Institute of Cardiovascular Science & Medicine.
|Awardees||Award Date||Honours / Awards / Prizes||Category|
|2002-12-01||Faculty Teaching Medal: Faculty of Medicine, The University of Hong Kong||Teaching Accomplishment|
|Term Period||Position||Professional Societies|
|12/1996 - 11/1997||Member||The Institute of Cardiovascular Science & Medicine|
|12/1997 - 11/1999||Council Member||The Institute of Cardiovascular Science & Medicine|
|12/1999 - 11/2003||Honorary Secretary||The Institute of Cardiovascular Science & Medicine|
|12/2003 - 11/2005||Council Member||The Institute of Cardiovascular Science & Medicine|
|1991 -||Member||The Physiological Society (UK)|
|1991 -||Member||The Hong Kong Association of Sports Medicine & Sports Science|
|1995 -||Member||The American Physiological Society|
|1985-2004||Member||The Biochemical Society (UK)|
|Our research is focused on the mechanisms that regulate blood flow, particularly in the skeletal muscle circulation, which undergoes large flow changes in the transition from rest to exercise. We also study the interaction between central and peripheral control mechanisms of flow regulation in situations such as exercise or systemic hypoxia.||Control of skeletal muscle blood flow|
|We have shown that ATP release from skeletal muscle is dependent on activation of the cystic fibrosis transmembrane conductance regulator (CFTR): exercise lowers the intracellular pH of muscle cells, and pH-lowering elevates the intracellular cAMP concentration, resulting in Protein-Kinase-A-dependent activation of CFTR. However, ATP is not released through CFTR itself: CFTR interacts with other proteins in the muscle cell membrane so that CFTR activation brings about the opening of a separate ATP release pore.||ATP and adenosine release in skeletal muscle|
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